I draw crappy comics all the time, but can I actually write?
Well, I’m going to take this game I was tagged in by the lovely kindredspirit23 and let you be the judge. Being blog-tagged sure is fun! (not sarcasm)
Anyway, here’s the rules:
1) Go to page 7 or 77 of your current WIP.
2) Count down 7 lines.
3) Quote the next 7 lines (finish the thought, if you need to)
4) Choose 7 more authors to play
As I mentioned earlier, I’m not exactly a writer. I have no awesome novels-in-progress at the moment. Kindredspirit23 suggested that I use a school paper, since I have definitely written papers over 7 pages before.
And so I have. But none of them are saved on my current computer.
Well, unless I double-space the paper I wrote for my Advanced Writing class on the misregulation of lncRNA in cancer cells. It’s single-spaced, 5 pages right now– but if I double space it, we’ll be smack-dab in the Works Cited section!
My 7 lines: (or, more accurately, 7 and a half on the original Word document, since I had to finish that last citation there. Knowing the page numbers are important!)
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Hutchinson AD, Hosking JR, Kichenadasse G, Mattiske JK, and Wilson C. 2012. Objective and subjective cognitive impairment following chemotherapy for cancer: A systemic review. Cancer Treatment Reviews. 38(7): 926-934. DOI: 10.1016/j.ctrv.2012.05.002.
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Kaida D, Scheider-Poetsch T, Yoshida M. 2012. Splicing in oncogenesis and tumor suppression. Cancer Science. 103(9): 1611-1616. DOI: 10.1111/j.1349-7006.2012.02356.x.
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Xi LF, Schiffman M, Koutsky LA, Hulbert A, Lee SK, DeFilippis, V, Shen ZP, and Kiviat NB. 2012. Association of human papillomavirus type 31 variants with risk of cervical intraepithelial neoplasia grades 2-3. International Journal of Cancer. 131(10): 2300-2307. DOI: 10.1002/ijc.27520.
Okay, okay. That’s a dick move.
I’m going to do this properly. I’m going to triple-space my essay to get us 7 lines of actual content: (or, in this case, 6.5 lines on the original Word document)
“miR-31 then suppresses several metastasis-promoting genes, including RhoA and WAVE3, which are linked to p53. LOC554202, in this case, is only a part of a long chain of gene interactions, which could be detrimental when developing TNCB treatments based on LOC554202. For instance, LOC554202 and miR-31 appear to be regulated epigenetically via CpG island hypermethylation. When researchers altered the DNA methylation in an attempt to upregulate LOC554202 and miR-31, the miR-31 levels did not reach those of the lncRNA host transcript. This could possibly be attributed to other factors regulating mi-R31, making LOC554202 an ineffective target for therapy.”
Yeah.
There’s a reason I draw instead of write.
(Thanks again to kindredspirit23 for tagging me! This was very entertaining to write!)