I draw crappy comics all the time, but can I actually write?
Anyway, here’s the rules:
1) Go to page 7 or 77 of your current WIP.
2) Count down 7 lines.
3) Quote the next 7 lines (finish the thought, if you need to)
4) Choose 7 more authors to play
As I mentioned earlier, I’m not exactly a writer. I have no awesome novels-in-progress at the moment. Kindredspirit23 suggested that I use a school paper, since I have definitely written papers over 7 pages before.
And so I have. But none of them are saved on my current computer.
Well, unless I double-space the paper I wrote for my Advanced Writing class on the misregulation of lncRNA in cancer cells. It’s single-spaced, 5 pages right now– but if I double space it, we’ll be smack-dab in the Works Cited section!
My 7 lines: (or, more accurately, 7 and a half on the original Word document, since I had to finish that last citation there. Knowing the page numbers are important!)
Hutchinson AD, Hosking JR, Kichenadasse G, Mattiske JK, and Wilson C. 2012. Objective and subjective cognitive impairment following chemotherapy for cancer: A systemic review. Cancer Treatment Reviews. 38(7): 926-934. DOI: 10.1016/j.ctrv.2012.05.002.
Kaida D, Scheider-Poetsch T, Yoshida M. 2012. Splicing in oncogenesis and tumor suppression. Cancer Science. 103(9): 1611-1616. DOI: 10.1111/j.1349-7006.2012.02356.x.
Xi LF, Schiffman M, Koutsky LA, Hulbert A, Lee SK, DeFilippis, V, Shen ZP, and Kiviat NB. 2012. Association of human papillomavirus type 31 variants with risk of cervical intraepithelial neoplasia grades 2-3. International Journal of Cancer. 131(10): 2300-2307. DOI: 10.1002/ijc.27520.
Okay, okay. That’s a dick move.
I’m going to do this properly. I’m going to triple-space my essay to get us 7 lines of actual content: (or, in this case, 6.5 lines on the original Word document)
“miR-31 then suppresses several metastasis-promoting genes, including RhoA and WAVE3, which are linked to p53. LOC554202, in this case, is only a part of a long chain of gene interactions, which could be detrimental when developing TNCB treatments based on LOC554202. For instance, LOC554202 and miR-31 appear to be regulated epigenetically via CpG island hypermethylation. When researchers altered the DNA methylation in an attempt to upregulate LOC554202 and miR-31, the miR-31 levels did not reach those of the lncRNA host transcript. This could possibly be attributed to other factors regulating mi-R31, making LOC554202 an ineffective target for therapy.”
There’s a reason I draw instead of write.
(Thanks again to kindredspirit23 for tagging me! This was very entertaining to write!)